[1]Ishida Y, Agata Y, ShibaharaK, et al. Induced expression of PD-1,a novel member of the immunoglobulin gene superfamily, upon programmed celldeath [J]. EMBO J, 1992,11(11):3887-2895.
[2] Okazaki T, Wang J. PD-1/PD-L pathway and autoimmunity[J].Autoimmunity, 2005,38(5):353-357.
[3] Okudaira K, Hokari R, Tsuzuki Y, etal. Blockade of B7-H1 or B7-DCinduces an anti-tumor effect in a mouse pancreatic cancer model [J].Int J Oncol,2009,35(4):741-749.
[4] Iwai Y, Ishida M, Tanaka Y, etal. Involvement of PD-L1 ontumor cells in the escape from host immune system and tumor immunotherapy byPD-L1 blockade [J]. Proc Natl Acad Sci USA, 2002,99(19):12293-12297.
[5] Dulos J, Carven GJ, van Boxtel SJ, et al. PD-1 blockade augments Th1 and Th17 and suppresses Th2 responses inperipheral blood from patients with prostate and advanced melanoma cancer [J]. J Immunother,2012,35(2):169-178.
[6] Monney L, Sabatos CA, Gaglia JL, etal. Th1-specific cell surface protein Tim-3 regulates macrophage activationand severity of an autoimmune diease [J].Nature, 2002,415(6871):536-541.
[7] Aaskura H, Kashio Y, Nakamura K, etal. Selective eosinophil adhesion to fibroblast via IFN-gamma-inducedgalectin-9 [J]. Immunol, 2002,169(10):5912-5918.
[8] Kaori S, Pushpa J, Samuel M, etal. Emerging Tim-3 functions in anti-microbial and tumor immunity [J].Trends Immunol, 2011,32(8):345-349.
如果感兴趣可以上华美生物官网(www.cusabio.cn)了解产品详情!