API 30-32006-1982
正己烷对大脑和周围神经诱发反应的浓度相关影响

CONCENTRATION-RELATED EFFECTS OF HEXANE ON EVOKED RESPONESES FROM BRAIN AND PERIPHERAL NERVE

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标准号

API 30-32006-1982

发布

1982年

发布单位

API - American Petroleum Institute

当前最新

API 30-32006-1982

 

 

适用范围

Introduction n-Hexane produces@ by way of its metabolite 2@S-hexanedione@ peripheral and central axonopathies with secondary myelinopathies [ 1 ]. Extensive ncuromorphologic investigations have described the anatomic changes associated with hexane exposures [2. 3]@ but there have been few studies of the functional@ electrophysiologic@ consequences of hexacarbon neuropathy. Of these studies@ most have focused on the peripheral nervous system@ even though the central nervous system (CNS) is known to undergo morphologic changes |4]. Ciancetti el al. |5J@ for example@ observed electromyographic and nerve conduction velocity changes in a large number of exposed shoe-industry workers@ and several investigators have described similar changes in animals with experimentally induced hexacarbon polyneuropathy [6@ 7]. In an earlier study (^ we showed@ by measuring the brainstem auditory-evoked response (BAER) and action potential (AP) of the ventral caudal nerve of the rat's tail@ that hexane causes a nerve conduction delay in auditory brainstem tracts as well as in peripheral nerve. This finding@ and a report that n-hexane causes pathology in the hypothalamus and optic tracts of cats [4]@ suggest that manifestations of hexane poisoning would be evident in evoked responses obtained from several sensory systems. Indeed@ Seppatainen et al. [9] found@ that n-hexane Induced changes in the visual-evoked response (VER) of exposed workers. In order to characterize further the electrophysiologic concomitants of hexacarbon neuropathy@ biopotentials?voked in peripheral nerve and brain by somatosensory@ auditory@ and visual stimulation were studied in rats exposed by inhalation to three concentrations of hexane.

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